Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Alternatively, our capability to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by greater pFAK localization in five-HT synapses. Our https://johnn421lxi1.blog-gold.com/profile